If you could somehow stimulate production of this enzyme, you might be able to render animals, and perhaps humans, resistant to high-fat diet-induced diabetes.

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It may be that suppressing insulin production to some degree could be beneficial in such disorders.

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This finding suggests new approaches to the prevention and treatment of diabetes.

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Our findings suggest that the current human epidemic in type-2 diabetes may be a result of GnT-4a enzyme deficiency.

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